Liver cirrhosis is often accompanied by cardiovascular lesions, including hyperdynamic circulatory states and myocardial lesions. The hyperdynamic circulatory state manifests as increased cardiac output, peripheral vasodilation, and decreased mean blood pressure. Cirrhotic cardiomyopathy involves impaired cardiac systolic and/or diastolic function, ventricular cavity enlargement or ventricular hypertrophy, and electrophysiological abnormalities. The pathogenesis of cirrhotic cardiomyopathy remains unclear, but gases such as nitric oxide, inflammatory factors, oxidative stress systems, apoptotic factors, sympathetic and parasympathetic nervous systems, endocannabinoid systems, and ion channels all play a role. The treatment of cirrhotic cardiomyopathy is primarily limited to animal studies; however, albumin has clinical therapeutic effectiveness. 肝硬化经常合并心血管病变，包括高动力循环状态和心肌病变。高动力循环状态表现为心输出量增加、周围血管扩张及平均血压降低。肝硬化心肌病包括心脏收缩和/或舒张功能障碍，心室腔增大或心室肥厚、电生理异常。肝硬化心肌病的发病机制仍不清楚，一氧化氮等气体、炎性因子、氧化应激系统、凋亡因子、交感和副交感神经系统、内源性大麻素系统以及离子通道等都起到一定作用。肝硬化心肌病的治疗多限于动物实验阶段，白蛋白有临床治疗效果。.