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Red Cell Distribution Width-Standard Deviation and the Severity of In-Stent Restenosis: Associations with Angiographic Stenosis Burden and Mehran Classification.

Red Cell Distribution Width-Standard Deviation and the Severity of In-Stent Restenosis: Associations with Angiographic Stenosis Burden and Mehran Classification.

期刊: Medicina (Kaunas, Lithuania) 日期: 2026-07-14 PMID: 42512900 DOI: 10.3390/medicina62071358 浏览: 25
作者: Savcilioglu MD, Lule KO, Buyukcelebi O, Vuruskan E
MD, S., KO, L., O, B., & E, V. (2026). Red Cell Distribution Width-Standard Deviation and the Severity of In-Stent Restenosis: Associations with Angiographic Stenosis Burden and Mehran Classification.. Medicina (Kaunas, Lithuania). https://doi.org/10.3390/medicina62071358
MD S, KO L, O B, E V. Red Cell Distribution Width-Standard Deviation and the Severity of In-Stent Restenosis: Associations with Angiographic Stenosis Burden and Mehran Classification.. Medicina (Kaunas, Lithuania). 2026; doi: 10.3390/medicina62071358
MD S, KO L, O B, et al. Red Cell Distribution Width-Standard Deviation and the Severity of In-Stent Restenosis: Associations with Angiographic Stenosis Burden and Mehran Classification.[J]. Medicina (Kaunas, Lithuania). 2026. DOI: 10.3390/medicina62071358.
@article{md2026,
  author = {Savcilioglu MD and Lule KO and Buyukcelebi O and Vuruskan E},
  title = {Red Cell Distribution Width-Standard Deviation and the Severity of In-Stent Restenosis: Associations with Angiographic Stenosis Burden and Mehran Classification.},
  journal = {Medicina (Kaunas, Lithuania)},
  year = {2026},
  doi = {10.3390/medicina62071358},
  note = {PMID: 42512900},
}
TY  - JOUR
AU  - Savcilioglu MD
AU  - Lule KO
AU  - Buyukcelebi O
AU  - Vuruskan E
TI  - Red Cell Distribution Width-Standard Deviation and the Severity of In-Stent Restenosis: Associations with Angiographic Stenosis Burden and Mehran Classification.
T2  - Medicina (Kaunas, Lithuania)
PY  - 2026
DO  - 10.3390/medicina62071358
AN  - PMID:42512900
ER  - 

摘要

Background and Objectives: Red cell distribution width-standard deviation (RDW-SD) has been associated with systemic inflammation and adverse cardiovascular outcomes, but its relationship with the angiographic severity and morphological complexity of drug-eluting stent-in-stent restenosis (ISR) has not been systematically characterized. The present study investigated whether RDW-SD is associated with angiographic restenosis severity and restenotic lesion complexity, and compared its performance with the platelet distribution width (PDW), Metabolic Stress Index (MSI), and Platelet-to-HDL Ratio (PHR). Materials and Methods: In this retrospective single-center observational study, 290 patients undergoing clinically indicated repeat coronary angiography following prior drug-eluting stent (DES) implantation were enrolled. Angiographic luminal narrowing was quantified by QCA and categorized as reference (<50% in-stent luminal narrowing; n = 111), intermediate ISR (50-69%; n = 76), and severe ISR (≥70%; n = 103). The Mehran classification was applied to patients with ISR ≥50% and dichotomized as Mehran class I-II (n = 91) vs. Mehran class III-IV (n = 70). Multivariable logistic regression, hierarchical modeling, and incremental discrimination analyses (IDI and NRI) were performed for both binary outcomes. Results: RDW-SD differed significantly across angiographic severity groups (Kruskal-Wallis H = 51.14, p < 0.001), being highest in the ISR ≥70% group [44.6 fL (IQR 43.8-45.3)] and lowest in the ISR 50-69% group [43.2 fL (42.7-43.7)]. A parallel pattern was observed across Mehran class (H = 50.57, p < 0.001; Mehran class III-IV: 44.9 fL [44.2-45.8]). In multivariable analysis, RDW-SD independently associated with ISR ≥70% (OR = 1.228 per 0.5 fL, 95% CI 1.122-1.344, p < 0.001) and Mehran class III-IV (OR = 1.274, 95% CI 1.155-1.406, p < 0.001). Hierarchical modeling showed that adding RDW-SD improved the AUC from 0.603 to 0.719 for ISR ≥ 70% and from 0.592 to 0.757 for Mehran class III-IV (LRT p < 0.001 for both), with incrementally larger IDI and NRI gains for the Mehran class III-IV outcome. PDW did not retain significance after adjustment; MSI and PHR were not significantly associated with either outcome. Conclusions: RDW-SD was independently associated with both angiographic ISR severity and Mehran morphological complexity in patients with established drug-eluting stent restenosis, with numerically greater model discrimination for the Mehran class III-IV endpoint. These findings suggest that RDW-SD may provide complementary information regarding restenosis burden and complexity in patients with established ISR. Prospective studies are required to validate these observations and determine their clinical relevance.

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