Giri, Suo, Pardi, Fishbein, Rezvani, Chen, Cui, & Wang (2026). Nuclear export by COPS5/CSN5/JAB1 mediates vascular smooth muscle cell dedifferentiation in neointimal hyperplasia.. Cardiovascular research.
Giri, Suo, Pardi, Fishbein, Rezvani, Chen, et al. Nuclear export by COPS5/CSN5/JAB1 mediates vascular smooth muscle cell dedifferentiation in neointimal hyperplasia.. Cardiovascular research. 2026; PMID: 41861053
Giri, Suo, Pardi, et al. Nuclear export by COPS5/CSN5/JAB1 mediates vascular smooth muscle cell dedifferentiation in neointimal hyperplasia.[J]. Cardiovascular research. 2026.
@article{giri2026,
author = {Giri and Suo and Pardi and Fishbein and Rezvani and Chen and Cui and Wang},
title = {Nuclear export by COPS5/CSN5/JAB1 mediates vascular smooth muscle cell dedifferentiation in neointimal hyperplasia.},
journal = {Cardiovascular research},
year = {2026},
note = {PMID: 41861053},
}
TY - JOUR AU - Giri AU - Suo AU - Pardi AU - Fishbein AU - Rezvani AU - Chen AU - Cui AU - Wang TI - Nuclear export by COPS5/CSN5/JAB1 mediates vascular smooth muscle cell dedifferentiation in neointimal hyperplasia. T2 - Cardiovascular research PY - 2026 AN - PMID:41861053 ER -
Vascular smooth muscle cell (VSMC) phenotypic remodelling is pivotal to neointimal hyperplasia (NH), a common pathological response to vascular injury. COPS5/CSN5/JAB1 harbours the deneddylase of the COP9 signalosome (CSN) holocomplex but also has deneddylation-independent function. The role of COPS5 in VSMCs remains obscure.