Anonymous (2026). LATS1/2-CD38 Metabolic Rewiring Links Senescence to Intraplaque Thrombosis.. Circulation research.
Anonymous. LATS1/2-CD38 Metabolic Rewiring Links Senescence to Intraplaque Thrombosis.. Circulation research. 2026; PMID: 42200276
Anonymous. LATS1/2-CD38 Metabolic Rewiring Links Senescence to Intraplaque Thrombosis.[J]. Circulation research. 2026.
@article{paper2026,
author = {Anonymous},
title = {LATS1/2-CD38 Metabolic Rewiring Links Senescence to Intraplaque Thrombosis.},
journal = {Circulation research},
year = {2026},
note = {PMID: 42200276},
}
TY - JOUR AU - Anonymous TI - LATS1/2-CD38 Metabolic Rewiring Links Senescence to Intraplaque Thrombosis. T2 - Circulation research PY - 2026 AN - PMID:42200276 ER -
Atherothrombosis, which underlies most acute coronary syndromes and is driven by intraplaque thrombosis, preferentially occurs in regions of disturbed blood flow (d-flow). Although LATS1/2 (large tumor suppressor kinases 1 and 2) are known regulators of endothelial mechanotransduction, the mechanisms by which d-flow connects endothelial senescence, proliferation, and intraplaque thrombosis remain poorly understood.