Anonymous (2026). GLI1 activation induced by intermittent hypoxia drives cardiac fibroblast activation via enhanced PKM2-mediated glycolysis.. European heart journal.
Anonymous. GLI1 activation induced by intermittent hypoxia drives cardiac fibroblast activation via enhanced PKM2-mediated glycolysis.. European heart journal. 2026; PMID: 42223116
Anonymous. GLI1 activation induced by intermittent hypoxia drives cardiac fibroblast activation via enhanced PKM2-mediated glycolysis.[J]. European heart journal. 2026.
@article{paper2026,
author = {Anonymous},
title = {GLI1 activation induced by intermittent hypoxia drives cardiac fibroblast activation via enhanced PKM2-mediated glycolysis.},
journal = {European heart journal},
year = {2026},
note = {PMID: 42223116},
}
TY - JOUR AU - Anonymous TI - GLI1 activation induced by intermittent hypoxia drives cardiac fibroblast activation via enhanced PKM2-mediated glycolysis. T2 - European heart journal PY - 2026 AN - PMID:42223116 ER -
Cardiac fibroblast (CF) activation in response to injury or inflammation is a central driver of pathological cardiac fibrosis, contributing to arrhythmias and heart failure. As previously demonstrated, intermittent hypoxia (IH), the hallmark pathophysiological feature of obstructive sleep apnoea (OSA), induces cardiac fibrosis. However, the mechanisms by which IH promotes CF activation and the key molecular mediators involved remain unclear.