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Ventricular Tachycardia Ablation in a Case of Calcified Left Ventricular Apical Aneurysm.

Ventricular Tachycardia Ablation in a Case of Calcified Left Ventricular Apical Aneurysm.

期刊: Cardiac electrophysiology clinics 日期: 2026-09-01 PMID: 42580793 DOI: 10.1016/j.ccep.2026.06.006 浏览: 7
作者: Adhikari U, Marimuthu S, Raja DC
U, A., S, M., & DC, R. (2026). Ventricular Tachycardia Ablation in a Case of Calcified Left Ventricular Apical Aneurysm.. Cardiac electrophysiology clinics. https://doi.org/10.1016/j.ccep.2026.06.006
U A, S M, DC R. Ventricular Tachycardia Ablation in a Case of Calcified Left Ventricular Apical Aneurysm.. Cardiac electrophysiology clinics. 2026; doi: 10.1016/j.ccep.2026.06.006
U A, S M, DC R. Ventricular Tachycardia Ablation in a Case of Calcified Left Ventricular Apical Aneurysm.[J]. Cardiac electrophysiology clinics. 2026. DOI: 10.1016/j.ccep.2026.06.006.
@article{u2026,
  author = {Adhikari U and Marimuthu S and Raja DC},
  title = {Ventricular Tachycardia Ablation in a Case of Calcified Left Ventricular Apical Aneurysm.},
  journal = {Cardiac electrophysiology clinics},
  year = {2026},
  doi = {10.1016/j.ccep.2026.06.006},
  note = {PMID: 42580793},
}
TY  - JOUR
AU  - Adhikari U
AU  - Marimuthu S
AU  - Raja DC
TI  - Ventricular Tachycardia Ablation in a Case of Calcified Left Ventricular Apical Aneurysm.
T2  - Cardiac electrophysiology clinics
PY  - 2026
DO  - 10.1016/j.ccep.2026.06.006
AN  - PMID:42580793
ER  - 

摘要

Calcific left ventricular (LV) apical aneurysms represent a highly arrhythmogenic substrate for ventricular tachycardias (VTs), posing unique challenges for mapping and catheter ablation. Dense calcification may obscure electroanatomic signals, hinder catheter stability, and alter conduction channels within scar tissue. We report a case of antiarrhythmic drug-refractory scar VT arising from a calcified LV apical aneurysm successfully treated using intracardiac echocardiogram (ICE) and 3-dimensional electroanatomic mapping guided catheter ablation. High-density substrate mapping revealed fragmented late potentials bordering calcified zones, identifying critical isthmus channels. Targeted ablation under fluoroscopy and ICE successfully terminated the VT and resulted in its noninducibility.

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