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In vitro multi-organ invasion model for exploring cardiac resistance to cancer metastasis.

In vitro multi-organ invasion model for exploring cardiac resistance to cancer metastasis.

期刊: Nature communications 日期: 2026-07-22 PMID: 42637759 DOI: 10.1038/s41467-026-75833-9 浏览: 10
作者: Shakeri A, Abdo D, Lei MHC, Okhovatian S, Beeghly GF, Popovic AM, Jiang R, Liu C, Wagner KT, Vosoughi D
A, S., D, A., MHC, L., S, O., GF, B., AM, P., R, J., C, L., KT, W., & D, V. (2026). In vitro multi-organ invasion model for exploring cardiac resistance to cancer metastasis.. Nature communications. https://doi.org/10.1038/s41467-026-75833-9
A S, D A, MHC L, S O, GF B, AM P, et al. In vitro multi-organ invasion model for exploring cardiac resistance to cancer metastasis.. Nature communications. 2026; doi: 10.1038/s41467-026-75833-9
A S, D A, MHC L, et al. In vitro multi-organ invasion model for exploring cardiac resistance to cancer metastasis.[J]. Nature communications. 2026. DOI: 10.1038/s41467-026-75833-9.
@article{a2026,
  author = {Shakeri A and Abdo D and Lei MHC and Okhovatian S and Beeghly GF and Popovic AM and Jiang R and Liu C and Wagner KT and Vosoughi D},
  title = {In vitro multi-organ invasion model for exploring cardiac resistance to cancer metastasis.},
  journal = {Nature communications},
  year = {2026},
  doi = {10.1038/s41467-026-75833-9},
  note = {PMID: 42637759},
}
TY  - JOUR
AU  - Shakeri A
AU  - Abdo D
AU  - Lei MHC
AU  - Okhovatian S
AU  - Beeghly GF
AU  - Popovic AM
AU  - Jiang R
AU  - Liu C
AU  - Wagner KT
AU  - Vosoughi D
TI  - In vitro multi-organ invasion model for exploring cardiac resistance to cancer metastasis.
T2  - Nature communications
PY  - 2026
DO  - 10.1038/s41467-026-75833-9
AN  - PMID:42637759
ER  - 

摘要

Despite the systemic spread of cancer, the ventricular myocardium is one of the least common sites of metastasis-a phenomenon that remains poorly understood. To examine this, we develop the Multi-organ Invasion Device (MInD), an organ-on-a-chip platform that enables multi-organ culture under flow. Organ compartments are connected in MInD using PermeoTubes-3D-printed porous conduits that support cancer cell intravasation, migration, and extravasation. In dual-organ devices, where highly aggressive breast cancer cells are co-cultured with either hepatic or cardiac tissue, invasion into cardiac tissue is significantly suppressed relative to hepatic co-culture. Importantly, in cardiac-hepatic-cancer tri-culture, the presence of cardiac tissue reduces overall invasion, with cancer cells preferentially migrating toward hepatic compartments. Cytokine profiling and RNA sequencing reveal that cardiac co-culture suppresses cell metastasis and invasion, while inducing immune activation. Overall, this platform presents an approach for uncovering organ-specific drivers of metastasis, accelerating future discovery of metastasis-inhibiting therapies.

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