Hypokalaemic paralysis as a presenting feature of primary aldosteronism.
S, K., V, K., M, A., N, R., & VR, D. (2026). Hypokalaemic paralysis as a presenting feature of primary aldosteronism.. BMJ case reports. https://doi.org/10.1136/bcr-2025-269580
S K, V K, M A, N R, VR D. Hypokalaemic paralysis as a presenting feature of primary aldosteronism.. BMJ case reports. 2026; doi: 10.1136/bcr-2025-269580
S K, V K, M A, et al. Hypokalaemic paralysis as a presenting feature of primary aldosteronism.[J]. BMJ case reports. 2026. DOI: 10.1136/bcr-2025-269580.
@article{s2026,
author = {Khurana S and Kumar V and Adil M and Raj N and Dubey VR},
title = {Hypokalaemic paralysis as a presenting feature of primary aldosteronism.},
journal = {BMJ case reports},
year = {2026},
doi = {10.1136/bcr-2025-269580},
note = {PMID: 42259571},
}
TY - JOUR AU - Khurana S AU - Kumar V AU - Adil M AU - Raj N AU - Dubey VR TI - Hypokalaemic paralysis as a presenting feature of primary aldosteronism. T2 - BMJ case reports PY - 2026 DO - 10.1136/bcr-2025-269580 AN - PMID:42259571 ER -
A woman in her 30s presented with acute flaccid paraparesis secondary to severe hypokalaemia. She had a known history of hypertension controlled on amlodipine and bisoprolol. Biochemical workup revealed metabolic alkalosis, renal potassium loss, suppressed plasma renin activity and markedly elevated aldosterone levels. CT imaging identified a left adrenal adenoma consistent with an aldosterone-producing adenoma. The patient underwent laparoscopic adrenalectomy, after which she no longer required antihypertensive medication and maintained normal serum potassium. This case highlights a reversible but often under-recognised cause of secondary hypertension presenting with hypokalaemic paraparesis.